Obesity as b-oxidation doesn't occur
Death in aerobic exercise
Death by hypothermia in cool conditions
Resistance to diet induced obesity
Malonyl-CoA
AMPK (AMP activated protein kinase)
ACC (Acetyl-CoA carboxylase
PKA (protein kinase A)
No ketoacyl-CoA transferase (converts acetoacetyl-CoA to Acetoacetate)
Can't pass the livers membrane
Produces hepatotoxic metabolites
No succinate CoA/succinate (esterifies acetoacetate to acetoacetyl-CoA)
Forms phosphatidyl molecules
Builds fatty acid chains
Builds glycerol molecules
Forms Farnesyl-CoA
Heart
Liver
Testicles
Lungs
Kidney
Muscle
Toxic to the hormone secreting glands
Impairs Hb ability to bind oxidation
Causes toxic lactic acid build up in tissues and organ
Close proximity of molecules involved allows for reactions to be coordinated
FAS is specifically attached to mitochondrial inner membranes
Coordinated regulation of expression due to having just 1 polypeptide to worry about
Efficiency is increased by intermediates being concentrated
Long chains of fatty acids
The fatty acid holding molecule is a lot less toxic
Stimulate higher levels of LDL
Lots of cis unsaturated bonds
Can be toxic in high amounts
Expensive to synthesise
Take a long time to synthesise
Cause acidosis
MACC
ACC
FAS
PACC
Glucokinase
Malonyl CoA
PKA
Farnesyl CoA
PKC
PKB
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