reduced by 5 years by the end of 2004.
reduced by 15 years by the year 2000.
reduced by 5 years by the year 2003.
reduced by 10 years by the year 2004.
remained at a plateau of 40,000 infections per year.
decreased to 20,000 per year.
increased, but at a slower rate of 100,000 per year.
decreased to less than 10,000 per year.
increased to 75,000 per year.
sexually transmitted infections.
it is determined largely during the initial phase of infection.
it has three distinct phases.
it takes more than15 years from infection to death in the absence of treatment.
it takes 8 to 12 years from infection to death in the absence of treatment.
it occurs in 60% to 70% of patients.
the response to antiretroviral therapy.
prospects for HIV eradication.
the risk for disease progression.
time from infection.
effect of HAART on the viral reservoirs in lymphoid tissue.
1 to 2 years.
5 to 6 years.
3 to 4 years.
10 years or more.
3 to 6 months.
Add photodynamic therapy.
Change to IV acyclovir.
Determine the patient’s HIV viral load and CD4 count.
Change to an alternative oral drug.
Obtain genital herpes culture and sensitivity testing.
clean intermittent catheterization.
standard pharmacologic measures.
checking viral load.
changing to an alternative protease inhibitor.
hydration, analgesics, and temporary cessation of indinavir.
noncontrast CT for diagnosis.
extracorporeal shockwave lithotripsy.
It seldom presents as renal insufficiency.
It may present as proteinuria.
It often responds to HAART.
It is more common in black patients.
Echogenic kidneys with preserved size are shown on ultrasonography.
has an incidence that has changed little since the advent of HAART.
is most common in patients who use intravenously administered drugs.
is related to coinfection with both HIV and a herpesvirus.
usually presents as renal disease.
is related to activation of epithelial cells.
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