reduced by 15 years by the year 2000.
reduced by 10 years by the year 2004.
reduced by 5 years by the year 2003.
reduced by 5 years by the end of 2004.
increased to 75,000 per year.
remained at a plateau of 40,000 infections per year.
increased, but at a slower rate of 100,000 per year.
decreased to less than 10,000 per year.
decreased to 20,000 per year.
sexually transmitted infections.
it has three distinct phases.
it takes more than15 years from infection to death in the absence of treatment.
it takes 8 to 12 years from infection to death in the absence of treatment.
it is determined largely during the initial phase of infection.
it occurs in 60% to 70% of patients.
the response to antiretroviral therapy.
prospects for HIV eradication.
the risk for disease progression.
effect of HAART on the viral reservoirs in lymphoid tissue.
time from infection.
10 years or more.
1 to 2 years.
3 to 6 months.
3 to 4 years.
5 to 6 years.
Determine the patient’s HIV viral load and CD4 count.
Obtain genital herpes culture and sensitivity testing.
Add photodynamic therapy.
Change to IV acyclovir.
Change to an alternative oral drug.
clean intermittent catheterization.
standard pharmacologic measures.
checking viral load.
hydration, analgesics, and temporary cessation of indinavir.
extracorporeal shockwave lithotripsy.
noncontrast CT for diagnosis.
changing to an alternative protease inhibitor.
It may present as proteinuria.
Echogenic kidneys with preserved size are shown on ultrasonography.
It seldom presents as renal insufficiency.
It often responds to HAART.
It is more common in black patients.
has an incidence that has changed little since the advent of HAART.
is related to coinfection with both HIV and a herpesvirus.
is related to activation of epithelial cells.
is most common in patients who use intravenously administered drugs.
usually presents as renal disease.
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