reduced by 10 years by the year 2004.
reduced by 5 years by the year 2003.
reduced by 5 years by the end of 2004.
reduced by 15 years by the year 2000.
increased, but at a slower rate of 100,000 per year.
remained at a plateau of 40,000 infections per year.
decreased to 20,000 per year.
increased to 75,000 per year.
decreased to less than 10,000 per year.
sexually transmitted infections.
it takes 8 to 12 years from infection to death in the absence of treatment.
it is determined largely during the initial phase of infection.
it has three distinct phases.
it occurs in 60% to 70% of patients.
it takes more than15 years from infection to death in the absence of treatment.
the response to antiretroviral therapy.
time from infection.
the risk for disease progression.
effect of HAART on the viral reservoirs in lymphoid tissue.
prospects for HIV eradication.
5 to 6 years.
3 to 4 years.
1 to 2 years.
3 to 6 months.
10 years or more.
Change to IV acyclovir.
Add photodynamic therapy.
Determine the patient’s HIV viral load and CD4 count.
Obtain genital herpes culture and sensitivity testing.
Change to an alternative oral drug.
checking viral load.
standard pharmacologic measures.
clean intermittent catheterization.
changing to an alternative protease inhibitor.
noncontrast CT for diagnosis.
extracorporeal shockwave lithotripsy.
hydration, analgesics, and temporary cessation of indinavir.
It is more common in black patients.
It may present as proteinuria.
It seldom presents as renal insufficiency.
It often responds to HAART.
Echogenic kidneys with preserved size are shown on ultrasonography.
usually presents as renal disease.
has an incidence that has changed little since the advent of HAART.
is most common in patients who use intravenously administered drugs.
is related to activation of epithelial cells.
is related to coinfection with both HIV and a herpesvirus.
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