reduced by 5 years by the end of 2004.
reduced by 15 years by the year 2000.
reduced by 10 years by the year 2004.
reduced by 5 years by the year 2003.
decreased to less than 10,000 per year.
increased to 75,000 per year.
remained at a plateau of 40,000 infections per year.
decreased to 20,000 per year.
increased, but at a slower rate of 100,000 per year.
sexually transmitted infections.
it has three distinct phases.
it takes 8 to 12 years from infection to death in the absence of treatment.
it occurs in 60% to 70% of patients.
it takes more than15 years from infection to death in the absence of treatment.
it is determined largely during the initial phase of infection.
prospects for HIV eradication.
the risk for disease progression.
effect of HAART on the viral reservoirs in lymphoid tissue.
time from infection.
the response to antiretroviral therapy.
1 to 2 years.
3 to 6 months.
10 years or more.
3 to 4 years.
5 to 6 years.
Add photodynamic therapy.
Change to IV acyclovir.
Determine the patient’s HIV viral load and CD4 count.
Change to an alternative oral drug.
Obtain genital herpes culture and sensitivity testing.
clean intermittent catheterization.
standard pharmacologic measures.
checking viral load.
changing to an alternative protease inhibitor.
noncontrast CT for diagnosis.
extracorporeal shockwave lithotripsy.
hydration, analgesics, and temporary cessation of indinavir.
It seldom presents as renal insufficiency.
It may present as proteinuria.
It often responds to HAART.
Echogenic kidneys with preserved size are shown on ultrasonography.
It is more common in black patients.
usually presents as renal disease.
has an incidence that has changed little since the advent of HAART.
is related to coinfection with both HIV and a herpesvirus.
is most common in patients who use intravenously administered drugs.
is related to activation of epithelial cells.
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