reduced by 15 years by the year 2000.
reduced by 10 years by the year 2004.
reduced by 5 years by the year 2003.
reduced by 5 years by the end of 2004.
remained at a plateau of 40,000 infections per year.
decreased to 20,000 per year.
increased to 75,000 per year.
increased, but at a slower rate of 100,000 per year.
decreased to less than 10,000 per year.
sexually transmitted infections.
it is determined largely during the initial phase of infection.
it occurs in 60% to 70% of patients.
it takes more than15 years from infection to death in the absence of treatment.
it has three distinct phases.
it takes 8 to 12 years from infection to death in the absence of treatment.
the risk for disease progression.
prospects for HIV eradication.
the response to antiretroviral therapy.
time from infection.
effect of HAART on the viral reservoirs in lymphoid tissue.
3 to 4 years.
3 to 6 months.
10 years or more.
5 to 6 years.
1 to 2 years.
Change to IV acyclovir.
Determine the patient’s HIV viral load and CD4 count.
Add photodynamic therapy.
Obtain genital herpes culture and sensitivity testing.
Change to an alternative oral drug.
standard pharmacologic measures.
clean intermittent catheterization.
checking viral load.
changing to an alternative protease inhibitor.
hydration, analgesics, and temporary cessation of indinavir.
extracorporeal shockwave lithotripsy.
noncontrast CT for diagnosis.
Echogenic kidneys with preserved size are shown on ultrasonography.
It is more common in black patients.
It may present as proteinuria.
It often responds to HAART.
It seldom presents as renal insufficiency.
has an incidence that has changed little since the advent of HAART.
is most common in patients who use intravenously administered drugs.
is related to activation of epithelial cells.
usually presents as renal disease.
is related to coinfection with both HIV and a herpesvirus.
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