reduced by 5 years by the year 2003.
reduced by 5 years by the end of 2004.
reduced by 10 years by the year 2004.
reduced by 15 years by the year 2000.
decreased to 20,000 per year.
remained at a plateau of 40,000 infections per year.
increased to 75,000 per year.
decreased to less than 10,000 per year.
increased, but at a slower rate of 100,000 per year.
sexually transmitted infections.
it is determined largely during the initial phase of infection.
it takes 8 to 12 years from infection to death in the absence of treatment.
it has three distinct phases.
it takes more than15 years from infection to death in the absence of treatment.
it occurs in 60% to 70% of patients.
the risk for disease progression.
prospects for HIV eradication.
effect of HAART on the viral reservoirs in lymphoid tissue.
the response to antiretroviral therapy.
time from infection.
3 to 4 years.
1 to 2 years.
10 years or more.
3 to 6 months.
5 to 6 years.
Determine the patient’s HIV viral load and CD4 count.
Obtain genital herpes culture and sensitivity testing.
Change to IV acyclovir.
Add photodynamic therapy.
Change to an alternative oral drug.
standard pharmacologic measures.
checking viral load.
clean intermittent catheterization.
extracorporeal shockwave lithotripsy.
noncontrast CT for diagnosis.
hydration, analgesics, and temporary cessation of indinavir.
changing to an alternative protease inhibitor.
It seldom presents as renal insufficiency.
It is more common in black patients.
It often responds to HAART.
It may present as proteinuria.
Echogenic kidneys with preserved size are shown on ultrasonography.
has an incidence that has changed little since the advent of HAART.
is most common in patients who use intravenously administered drugs.
usually presents as renal disease.
is related to coinfection with both HIV and a herpesvirus.
is related to activation of epithelial cells.
選擇要在Apple App Store上查看的Topgrade應用程序。