Incomplete bladder emptying

A contraction of reduced strength

A contraction of reduced speed

Prolonged bladder emptying

A contraction of reduced duration

symptoms of impaired bladder emptying in the absence of bladder outlet obstruction.

reduced desire to void, associated with incomplete bladder emptying.

There is currently no recognized definition for UAB.

infrequent voiding, associated with voiding symptoms and increased postvoid residual.

reduced desire to void, usually accompanied by urinary frequency or nocturia, with or without incontinence that predominates at night.

Myelohypertrophy pattern

Degeneration pattern

Dysjunction pattern

Dense band pattern

Dysfunction pattern

Speed

Strength

Sustainability

Duration

Efficiency

Bladder contractility index

Watt factor

Detrusor contraction coefficient

Projected isovolumetric pressure

Detrusor contraction duration

Muscarinic agonists can cause severe cardiac depression.

Muscarinic agonists are more likely to be effective in patients with complete bladder denervation.

Muscarinic agonists show good efficacy in restoring contractility.

None of the above.

Anticholinesterases can cause uterine contraction.

None of the above

Stimulation of mechanoreceptive afferent nerves

Direct stimulation of detrusor myocytes

Inhibition of pathologic urethral afferent signaling

Direct stimulation of efferent nerves

Acontractile bladder

Myogenic etiology

Intact bladder sensation

Older age

Neurogenic etiology

Cerebrovascular accident—postacute phase

Brain tumor

Parkinson disease

Multisystem atrophy

Multiple sclerosis

Most will require bladder outlet surgery by 10 years

No studies are available

Deterioration in symptoms but not urodynamic parameters

No significant change in urodynamic parameters for at least 10 years

Improvement in contractility in at least 50% of individuals