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bio sem1

bio-sem1

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84113

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120

block

negatve feedback loop

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response levels increased
return to homeostsis
stress causing decrease of
controlled condition
receptors (input) increase
effectors increase
control centre (output) increase

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negative feedback loop of glucocorticoid

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controlled condition
receptors stimulate
control centre ant pituitory
effector zona fasiculata in adrenal cortex

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cells in hypothalumus to release CRH
increases ACTH output
decreased glucocorticoid level
secretes glucocorticoids

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homeostatic control

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control centre (CNS)
deviation
effector
normal limits
receptor
negative or positive feedback to control centre
reversal or enhancement

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an image is focused on the r__ stimulates p_ ,which transduce light stimulus into r_ p__ and pass information to b cells, inturn communicate with g_ cells, which project axons to the T_ ,fibers carrying visual nerve impulses to the p_ v__ c_

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84117

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deep wound healing

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Inflammatory phase
Migratory phase
Proliferative phase
Maturation phase

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epithelial cells grow beneath scab
blood clot forms, unites wound edges
scab falls off, once the epidermis is restored to nomal
scab (granulation tissue), damaged blood vessels regrow

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epiphyseal growth zones

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Proliferating cartilage zone, chondrocytes replace dead, diaphyseal surface /epiphyseal plate
Calified cartilage zone, osteoblasts cement border to diaphysis
Hypertrophic zone, maturation of cells, lengthwise expansion
Resting cartilage zone, ancor the epiphyseal plate to the epiphysis

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fracture and repair of bone

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Procallus, fibroblasts invade & produce collagen fibers to connect broke ends
Remodelling, osteoclasts replace dead portions, compact bone replaces spongy bone around perphery of fracture
Fracture heamatoma forms (6-8)hrs, phagocytes w/ osteoclsts remove traumtised tissue
Bony callus, trabeculae from osteoblasts, join living & dead parts of original bone

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