The resting potential
Two of the above are correct
The driving force
The equlibrium potential
The membrane potential
It is a trasmembrane protein
Two of the above are incorrect
The channel pore is selectively permeable to only K ions
It is an ATP-dependant enzyme
It is a multimeric protein consisting of several subunits
Yes, because the intracellular and extracellular ionic concentrations are different, and molecules always diffuse down their concentration gradients
No, because there is almost no driving force for K, and because there is almost no conductance for Na
Yes, because neurons have a resting potential, meaning that V does not = 0, and there are channels in the membrane, so g does not = 0
None of the above is correct
No, because there are no open channels and thus g=0
It uses specialized glial cells called astrocytes to remove the extra K
This is a trick question; excessive extracellular K does not affect neurons aversely
It yses K leak channels to allow the extra K leak out of the central nervous system
It uses ATP- dependent Na/K pumps to remove the extra K
K leak channel
Voltage-gated Ca channel
Voltage-gated Na channel
Voltage-gated (delayed rectifying) K channel
The membrane potential will rise, but if enough current is injected the membrane potential will spike. The frequency of the spikes will increase with increased current injection.
The membrane potential will rise, but if enough current is injected the membrane potential will spike. The amplitude of the spike will increase with increased current injection.
The membrane potential will proportionally get more and more postitive (or less and less negative)
The membrane potential will get more positive, but reach a plateau and no longer increase
None of the above are correct
During the undershoot phase, when the membrane is hyperpolarized lower than the resting potential.
During the rising phase when the membrane potential is rapidly increasing
During the overshoot phase when the membrane potential rises above 0 mV
When the membrane is at its resting potential
During the falling phase when the membrane potential becomes negative again
delayed rectifying K channel
voltage-gated Ca channel
voltage-gated Na channel
acetylcholine-gated cation channel
Intracellular Ca released from the endoplasmic reticulum
Intracellular Ca released from the synaptic vesicles
Intracellular Ca exiting the axon terminal through voltage-gated Ca channels
Extracellular Ca entering the axon terminal through voltage-gated Ca channels
Yes, it can sometimes act through the shortcut pathway wherein activated heterotrimeric G proteins directly affect ion channel activity
Yes, sometimes by directly causing a channel pore to open in the receptor, allowing ions to flow through
None of the above is incorrect
No; metabotropic receptors always act by triggering gene transcription and therefore are signifcantly slwoer than ionotropic receptors
No; metabotropic receptors always act through second messengers and therefore are significantly slower than ionotropic receptors by minutes
A larger synapse will likely be more resistant to neurotoxins
A large synapse will likely trigger an action potential with larger amplitude
A large synapse will likely contain more synaptic vesicles, and therefore lead to more neurotransmitter release
A large synapse will likely contain more postsynaptic receptors, and therefore have a greater postsynaptic response to neurotransmitter release
The molecule is synthesized and stored in the presynaptic neuron
None of the aboe is correct
The receptor for the neurotransmitter is expressed in the postsynaptic neuron
The molecule is secreted by the presynaptic axon terminal
The molecule generates a postsynaptic response similar to when the presynaptic neuron is stimulated
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